There is ample evidence that obesity is important in the etiology of hypertension. Body composition such as % fat, WHR, BMI are the most common indexes of obesity assesment used I studies of obesity. WHR is higher in hypertensive obesity and
obesity
than in hypertension and normotensive subjects.
Other methods to examine cardiovascular function, such as heart rate, blood pressure, myocardial oxygen uptake, oxygen uptake per body weight response to treadmill exercise testing have all provided further evidence for obesity in hypertension,
especially in middle aged subjects.
Group with hypertensive obesity have decreased heart rate in the maximal exercise and during recovery time compared with the other groups. %TBW is significantly lower in hypertensive obesity and obesity group than hypertension and normotensive
group(p<0.05). Blood pressure is higher in hypertensive groups compared with non-hypertensive groups. But obesity group have increased blood pressure compared with normotensive subjects. At resting, hypertensive obesity was lower ST level than
hypertension and normotensive group. Especially, t recovery 5 minute, obesity group had 84% lower ST level (lead II) than normotensive group. MVO2 and oxygen uptake per body weight were significantly(p<0.05) negative affected by hypertension and
obesity. These results indicate that cardiovasculr dysfunction assosiated with hypertention and obesity.
|